Modulating mucins makes melanin

DC FieldValueLanguage
dc.contributor.authorCasalou, Cristina-
dc.contributor.authorTobin, Desmond John-
dc.date.accessioned2022-01-14T16:05:11Z-
dc.date.available2022-01-14T16:05:11Z-
dc.date.copyright2021 British Association of Dermatologistsen_US
dc.date.issued2021-11-24-
dc.identifier.citationBritish Journal of Dermatologyen_US
dc.identifier.issn0007-0963-
dc.identifier.urihttp://hdl.handle.net/10197/12732-
dc.description.abstractThe outermost layer of skin, the epidermis has evolved to provide a physical barrier of protection from harmful substances and invasion of pathogens. The skin barrier consists in part of highly glycosylated proteins called mucins and the epithelium. Epidermal homeostasis is maintained by the proliferation and differentiation of its basal layer keratinocytes that migrate to the superficial layer of the epidermis, and through its regulated inter-cellular communication with the pigment producing cells, the melanocytes.en_US
dc.description.sponsorshipScience Foundation Irelanden_US
dc.format.mediumPrint-Electronic-
dc.language.isoenen_US
dc.publisherWileyen_US
dc.rightsThis is the peer reviewed version of the following article: British Journal of Dermatology, which has been published in final form at https://doi.org/10.1111/bjd.20877. This article may be used for non-commercial purposes in accordance with Wiley Terms and Conditions for Self-Archiving.en_US
dc.subjectDermatologyen_US
dc.subjectSkin pigmentationen_US
dc.titleModulating mucins makes melaninen_US
dc.typeJournal Articleen_US
dc.internal.authorcontactotherdesmond.tobin@ucd.ieen_US
dc.statusPeer revieweden_US
dc.check.date2022-07-14-
dc.identifier.doi10.1111/bjd.20877-
dc.neeo.contributorCasalou|Cristina|aut|-
dc.neeo.contributorTobin|Desmond John|aut|-
dc.description.adminUpdate citation details during checkdate report - RORen_US
dc.date.updated2022-01-11T13:58:35Z-
dc.identifier.grantid19/FFP/6752-
dc.rights.licensehttps://creativecommons.org/licenses/by/3.0/ie/en_US
item.fulltextWith Fulltext-
item.grantfulltextopen-
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Medicine Research Collection
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